4 European Projects Found
Searched on 125080 European Projects
FINISHED
Heart OMics in AGEing (HOMAGE)
Start date: Feb 1, 2013, End date: Jan 31, 2019,
As more people survive into old age, the prevalence of heart failure (HF), one of the most common and debilitating diseases in older people, will rise still further. Delaying or preventing HF will have great benefit to those at personal risk, their families, society and the economy. HOMAGE aims to provide a biomarker (BM) approach that will a) help identify i. patients at high risk of developing ...
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22
Partners Participants
FINISHED
Systems Biology to Identify Molecular Targets for Vascular Disease Treatment (SYSVASC)
Start date: Feb 1, 2014, End date: Jan 31, 2018,
Asymptomatic vascular damage accumulates for years before patients are identified and subjected to therapeutic measures. The limited knowledge on early vascular disease pathophysiology is reflected in the lack of therapeutic options. SysVasc aims to overcome this limitation by mounting a comprehensive systems medicine approach to elucidate pathological mechanisms, which will yield molecular target ...
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19
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Markers for Sub-Clinical Cardiovascular Risk Assessemnt (EU-MASCARA)
Start date: Dec 1, 2011, End date: May 31, 2016,
EU-MASCARA is a collaborative project that aims to improve diagnosis of cardiovascular diseases and prediction of cardiovascular risk by analysing a panel of biomarkers. EU-MASCARA aims to examine genetic, proteomic and metabolomic markers together with markers of inflammation, oxidative stress and cardiac remodelling to study their incremental diagnostic and predictive value over and above existi ...
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16
Partners Participants
FINISHED
The European Network for Translational Research in Atrial Fibrillation (EUTRAF)
Start date: Nov 1, 2010, End date: Oct 31, 2015,
"The initiation and perpetuation of atrial fibrillation (AF) can be regarded as a complication of a progressive transformation of the structure and functional properties of the atria. This transformation is the result of complex and multiple changes at the molecular, cellular and organ levels which interact to form the basis for proarrhythmic mechanisms in AF. Numerous individual and environmental ...
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